diff --git a/R/TobLags.R b/R/TobLags.R
index b458998..525ca5a 100644
--- a/R/TobLags.R
+++ b/R/TobLags.R
@@ -5,8 +5,9 @@
 #' gradually has an effect on the relative risk of disease incidence over time (up to 40 years)
 #' since e.g. someone quit smoking
 #'
-#' All lag times are taken from a re-analysis of the Cancer prevention II study by Oza et al 2011 and Kontis et al 2014
-#' The values were sent to us by Kontis. Lags are smoothed functions over time describing the proportion of
+#' All lag times are taken from a re-analysis of the Cancer prevention II study by \insertCite{Oza2011;textual}{tobalcepi}
+#'  and \insertCite{Kontis2014;textual}{tobalcepi}.  
+#' The values were sent to us on request by Kontis. Lags are smoothed functions over time describing the proportion of
 #' the excess risk due to smoking that still remains.
 #'
 #' Kontis et al. re-analysed the change in risk after smoking in the ACS-CPS II study from Oza et al.,
@@ -23,8 +24,8 @@
 #' on diabetes risk occur within a few years, more similar to the CVDs than cancers.
 #'  Therefore, we used the CVD curve for diabetes." In-line with Kontis, we apply the rate of decline
 #'  in risk of CVD after quitting smoking to type 2 diabetes.
-#'   For other diseases, we assume that the relative risk reverts to 1 immediately after quitting
-#'   i.e. an immediate rather than a gradual decline in risk.
+#'   For all remaining conditions we apply the most conservative estimate available 
+#'   and assume that the decline in risk follows the cancer estimate provided by Kontis et al., as this has the slowest decline in risk.   
 #'
 #' @param disease_name Character - the name of the disease under consideration.
 #' @param n_years Integer - the number of years from 1 to n over which the effect of a change in
@@ -39,7 +40,11 @@
 #' @importFrom data.table := setDT setnames data.table
 #' 
 #' @export
-#'
+#' @references
+#' \insertRef{Kontis2014}{tobalcepi}  
+#' 
+#' \insertRef{Oza2011}{tobalcepi}  
+#' 
 #'
 #' @examples
 #'
@@ -63,12 +68,10 @@ TobLags <- function(
 
   copd_lags <- c("Chronic_obstructive_pulmonary_disease")
 
-  # For other conditions assume that the excess risk is zero 1 year after cessation
-
+  
   #################################
   # Specify the functional forms of the lags
-  # The numbers are taken from SAPM - Holmes et al. 2012
-
+  
   ##################
   # Set the default
 
diff --git a/docs/articles/Alcohol-attributable_diseases_and_dose-response_curves.pdf b/docs/articles/Alcohol-attributable_diseases_and_dose-response_curves.pdf
index f530b8d..4f18e6c 100644
Binary files a/docs/articles/Alcohol-attributable_diseases_and_dose-response_curves.pdf and b/docs/articles/Alcohol-attributable_diseases_and_dose-response_curves.pdf differ
diff --git a/docs/articles/Smoking_and_the_risks_of_adult_diseases.pdf b/docs/articles/Smoking_and_the_risks_of_adult_diseases.pdf
index 7e38ebd..6b32e4e 100644
Binary files a/docs/articles/Smoking_and_the_risks_of_adult_diseases.pdf and b/docs/articles/Smoking_and_the_risks_of_adult_diseases.pdf differ
diff --git a/docs/pkgdown.yml b/docs/pkgdown.yml
index e98f35d..744afd7 100644
--- a/docs/pkgdown.yml
+++ b/docs/pkgdown.yml
@@ -7,7 +7,7 @@ articles:
   PAF_methodology_report: PAF_methodology_report.html
   Smoking_and_the_risks_of_adult_diseases: Smoking_and_the_risks_of_adult_diseases.pdf
   tobalcepi: tobalcepi.html
-last_built: 2023-06-06T10:27Z
+last_built: 2023-06-06T14:51Z
 urls:
   reference: https://stapm.github.io/tobalcepi/reference
   article: https://stapm.github.io/tobalcepi/articles
diff --git a/docs/reference/TobLags.html b/docs/reference/TobLags.html
index 77d71d6..da538a7 100644
--- a/docs/reference/TobLags.html
+++ b/docs/reference/TobLags.html
@@ -127,8 +127,11 @@ <h2>Value</h2>
     </div>
     <div id="details">
     <h2>Details</h2>
-    <p>All lag times are taken from a re-analysis of the Cancer prevention II study by Oza et al 2011 and Kontis et al 2014
-The values were sent to us by Kontis. Lags are smoothed functions over time describing the proportion of
+    <p>All lag times are taken from a re-analysis of the Cancer prevention II study by Oza et al. (2011)
+
+ and Kontis et al. (2014)
+.  
+The values were sent to us on request by Kontis. Lags are smoothed functions over time describing the proportion of
 the excess risk due to smoking that still remains.</p>
 <p>Kontis et al. re-analysed the change in risk after smoking in the ACS-CPS II study from Oza et al.,
 producing three functions to describe the decline in risk after quitting for each of cancers, CVD and COPD.
@@ -143,8 +146,19 @@ <h2>Details</h2>
 on diabetes risk occur within a few years, more similar to the CVDs than cancers.
  Therefore, we used the CVD curve for diabetes." In-line with Kontis, we apply the rate of decline
  in risk of CVD after quitting smoking to type 2 diabetes.
-  For other diseases, we assume that the relative risk reverts to 1 immediately after quitting
-  i.e. an immediate rather than a gradual decline in risk.</p>
+  For all remaining conditions we apply the most conservative estimate available 
+  and assume that the decline in risk follows the cancer estimate provided by Kontis et al., as this has the slowest decline in risk.</p>
+    </div>
+    <div id="references">
+    <h2>References</h2>
+    <p>Kontis V, Mathers CD, Rehm J, Stevens GA, Shield KD, Bonita R, Riley LM, Poznyak V, Beaglehole R, Ezzati M (2014).
+“Contribution of six risk factors to achieving the 25× 25 non-communicable disease mortality reduction target: a modelling study.”
+<em>The Lancet</em>, <b>384</b>(9941), 427-437.
+ISSN 0140-6736.</p>
+<p>Oza S, Thun MJ, Henley SJ, Lopez AD, Ezzati M (2011).
+“How many deaths are attributable to smoking in the United States? Comparison of methods for estimating smoking-attributable mortality when smoking prevalence changes.”
+<em>Preventive medicine</em>, <b>52</b>(6), 428-433.
+ISSN 0091-7435.</p>
     </div>
 
     <div id="ref-examples">
diff --git a/man/TobLags.Rd b/man/TobLags.Rd
index 3329aaa..716398d 100644
--- a/man/TobLags.Rd
+++ b/man/TobLags.Rd
@@ -30,8 +30,9 @@ gradually has an effect on the relative risk of disease incidence over time (up
 since e.g. someone quit smoking
 }
 \details{
-All lag times are taken from a re-analysis of the Cancer prevention II study by Oza et al 2011 and Kontis et al 2014
-The values were sent to us by Kontis. Lags are smoothed functions over time describing the proportion of
+All lag times are taken from a re-analysis of the Cancer prevention II study by \insertCite{Oza2011;textual}{tobalcepi}
+ and \insertCite{Kontis2014;textual}{tobalcepi}.  
+The values were sent to us on request by Kontis. Lags are smoothed functions over time describing the proportion of
 the excess risk due to smoking that still remains.
 
 Kontis et al. re-analysed the change in risk after smoking in the ACS-CPS II study from Oza et al.,
@@ -48,11 +49,16 @@ CVD or COPD. Kontis et al. state that
 on diabetes risk occur within a few years, more similar to the CVDs than cancers.
  Therefore, we used the CVD curve for diabetes." In-line with Kontis, we apply the rate of decline
  in risk of CVD after quitting smoking to type 2 diabetes.
-  For other diseases, we assume that the relative risk reverts to 1 immediately after quitting
-  i.e. an immediate rather than a gradual decline in risk.
+  For all remaining conditions we apply the most conservative estimate available 
+  and assume that the decline in risk follows the cancer estimate provided by Kontis et al., as this has the slowest decline in risk.
 }
 \examples{
 
 TobLags("Pharynx")
 
 }
+\references{
+\insertRef{Kontis2014}{tobalcepi}  
+
+\insertRef{Oza2011}{tobalcepi}
+}